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Nevertheless, the cells had issues within the systems that normally remove reactive oxygen species from cells, reduced levels of mitochondrial superoxide dismutase (a protective enzyme against oxidative stress), and other factors that contribute to ongoing oxidative damage

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Of relevance, recent evidence indicates that SAM exerts anti-inflammatory effects by reducing the expression of the proinflammatory cytokine tumor necrosis factor-alpha (TNFα) and increasing the expression of the anti-inflammatory cytokine interleukin 10 (IL-10)